Then auricularia auricula is a natural anticoagulant health care product.
Hong Zhaoguang, an expert in health education, particularly emphasizes eating black fungus!
give up smoking
There is a nicotine substance in tobacco, which is very toxic to human body. After nicotine is inhaled into human body, it stimulates autonomic nerve, causes vasospasm, quickens heartbeat, raises blood pressure and blood cholesterol, thus accelerating arteriosclerosis.
Prevention of pre-onset of cerebral infarction
The risk factors of cerebral infarction, if not carefully prevented, will change from quantitative to qualitative, until the outbreak of cerebrovascular disease. Therefore, we must prevent cerebral infarction as early as possible and be alert to various signals before cerebral infarction. It can be divided into five aspects.
(a) changes in consciousness and mental state, such as drowsiness, that is, groggy sleep all day. Abnormal personality, becoming withdrawn, indifferent and restless, and some of them may have temporary loss of consciousness or mental decline. These manifestations are related to cerebral ischemia.
(2) Dyskinesia is characterized by sudden mouth tilt, difficulty in speaking, unclear pronunciation, aphasia or lack of expression. Dysphagia, weakness or activity failure, loss of grip, unsteady walking, sudden fall, and some may have limb convulsions.
(3) Sensory disorder is characterized by numbness of tongue, face, lips and limbs. Deaf ears, sense of rotation in vision.
(4) Headache, dizziness and headache are different from the past, and the severity of headache lasts for a long time.
(5) Disorders such as autonomic nerve: general weakness, sweating, low fever, chest tightness, palpitation, sudden snoring, vomiting and other autonomic nerve symptoms. A few patients have facial adhesion, retinal hemorrhage and nosebleed. If the above symptoms appear, you should seek medical attention in time, make a detailed examination, make a diagnosis and deal with them in time to avoid or delay the onset of cerebral infarction.
Section III Treatment and Rehabilitation Methods
Treatment of acute phase
(a) general treatment should be bed rest, head flat, oxygen when necessary. Generally speaking, blood pressure will not decrease unless it is extremely high. Turn over frequently, pay attention to the unobstructed respiratory tract, prevent respiratory tract and urinary tract infections, and prevent bedsores.
(2) Low molecular dextran 500 ml, intravenous drip every day for two weeks.
(3) The vasodilator intermittently inhaled mixed gas (6-7% carbon dioxide, 50-95% oxygen), 250 ml of 5% sodium bicarbonate, 65,438+0-2 times, and 65,438+000 mg papaverine into 250 ml of glucose solution, once a day, for two weeks. Vasodilators should only be used in the first 1-2 days or 3 weeks, otherwise brain theft syndrome may occur, which will aggravate the hypoxic damage of brain tissue.
(4) Anticoagulant therapy is as follows: heparin12,500 units are added with 5% glucose, normal saline or 10% glucose solution, and intravenous drip slowly (at a speed of 20 drops per minute for 24-48 hours). On the first day, anticoagulant drugs can be taken orally at the same time, such as neodicoumarin 300 mg, dicoumarin 100-200 mg and neoanticoagulant 4-8 mg).
(5) Traditional Chinese medicines for promoting blood circulation and removing blood stasis, such as Danshen, Chuanxiong, Pueraria lobata, Angelica sinensis, Carthamus tinctorius, etc. Optional.
(6) The surgical treatment of carotid artery occlusion can adopt embolectomy. For patients with transient ischemic attack and intracranial arterial system obstruction, superficial temporal artery and middle cerebral artery anastomosis can be performed on the affected side.
(7) New acupuncture, scalp acupuncture, laser or ultrasonic therapy are available.
Treatment of cerebral infarction in convalescence
In the recovery period of cerebral infarction, peripheral circulation improvers should be taken appropriately to reduce platelet aggregation, reduce blood lipid, nourish nerves, promote nerve cell metabolism, pay attention to blood pressure and other treatments, and pay attention to the treatment of primary diseases for coronary heart disease, rheumatic heart disease and diabetes (see cerebral hemorrhage for details).
Prevention of pre-onset of cerebral infarction
The risk factors of cerebral infarction, if not carefully prevented, will change from quantitative to qualitative, until the outbreak of cerebrovascular disease. Therefore, we must prevent cerebral infarction as early as possible and be alert to various signals before cerebral infarction. It can be divided into five aspects.
(a) changes in consciousness and mental state, such as drowsiness, that is, groggy sleep all day. Abnormal personality, becoming withdrawn, indifferent and restless, and some of them may have temporary loss of consciousness or mental decline. These manifestations are related to cerebral ischemia.
(2) Dyskinesia is characterized by sudden mouth tilt, difficulty in speaking, unclear pronunciation, aphasia or lack of expression. Dysphagia, weakness or activity failure, loss of grip, unsteady walking, sudden fall, and some may have limb convulsions.
(3) Sensory disorder is characterized by numbness of tongue, face, lips and limbs. Deaf ears, sense of rotation in vision.
(4) Headache, dizziness and headache are different from the past, and the severity of headache lasts for a long time.
(5) Disorders such as autonomic nerve: general weakness, sweating, low fever, chest tightness, palpitation, sudden snoring, vomiting and other autonomic nerve symptoms. A few patients have facial adhesion, retinal hemorrhage and nosebleed. If the above symptoms appear, you should seek medical attention in time, make a detailed examination, make a diagnosis and deal with them in time to avoid or delay the onset of cerebral infarction.
Section III Treatment and Rehabilitation Methods
Treatment of acute phase
(a) general treatment should be bed rest, head flat, oxygen when necessary. Generally speaking, blood pressure will not decrease unless it is extremely high. Turn over frequently, pay attention to the unobstructed respiratory tract, prevent respiratory tract and urinary tract infections, and prevent bedsores.
(2) Low molecular dextran 500 ml, intravenous drip every day for two weeks.
(3) The vasodilator intermittently inhaled mixed gas (6-7% carbon dioxide, 50-95% oxygen), 250 ml of 5% sodium bicarbonate, 65,438+0-2 times, and 65,438+000 mg papaverine into 250 ml of glucose solution, once a day, for two weeks. Vasodilators should only be used in the first 1-2 days or 3 weeks, otherwise brain theft syndrome may occur, which will aggravate the hypoxic damage of brain tissue.
(4) Anticoagulant therapy is as follows: heparin12,500 units are added with 5% glucose, normal saline or 10% glucose solution, and intravenous drip slowly (at a speed of 20 drops per minute for 24-48 hours). On the first day, anticoagulant drugs can be taken orally at the same time, such as neodicoumarin 300 mg, dicoumarin 100-200 mg and neoanticoagulant 4-8 mg).
(5) Traditional Chinese medicines for promoting blood circulation and removing blood stasis, such as Danshen, Chuanxiong, Pueraria lobata, Angelica sinensis, Carthamus tinctorius, etc. Optional.
(6) The surgical treatment of carotid artery occlusion can adopt embolectomy. For patients with transient ischemic attack and intracranial arterial system obstruction, superficial temporal artery and middle cerebral artery anastomosis can be performed on the affected side.
(7) New acupuncture, scalp acupuncture, laser or ultrasonic therapy are available.
Treatment of cerebral infarction in convalescence
In the recovery period of cerebral infarction, peripheral circulation improvers should be taken appropriately to reduce platelet aggregation, reduce blood lipid, nourish nerves, promote nerve cell metabolism, pay attention to blood pressure and other treatments, and pay attention to the treatment of primary diseases for coronary heart disease, rheumatic heart disease and diabetes (see cerebral hemorrhage for details).